Case Files Pathology Lange Case Files by Eugene Toy, Margaret Uthman, Edward Uthman, Earl Brown

By Eugene Toy, Margaret Uthman, Edward Uthman, Earl Brown

Greater than 50 scientific instances assist you ace pathology classes and the USMLE
The Case documents sequence is an award-winning studying approach confirmed to enhance examination rankings. This sequence allows you to research within the context of genuine sufferers rather than easily memorizing. Case documents: Pathology: moment variation provides 50+ medical instances with open-ended questions which weave simple technological know-how thoughts into the medical situation. each one case comprises a longer dialogue (including definitions and a pathophysiology discussion), key issues, and 3-5 USMLE-style comprehension questions. The authors are skilled academics from the college of Texas-Houston scientific college and East Tennessee country collage Quillen collage of medication.

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It undergoes conjugation with glucuronic acid in the hepatocyte to form conjugated or direct bilirubin, which is water-soluble and nontoxic, and then is excreted into the bile canaliculus. When liver disease causes jaundice, affected individuals usually have reflux of conjugated bilirubin into the blood, which causes the visible icterus when deposited in tissues, as well as the dark urine resulting from urinary excretion of elevated levels of water-soluble conjugated bilirubin. Figure 3-1. Microscopic image of liver cirrhosis.

There may be hepatocyte swelling called ballooning degeneration, as well as liver cell necrosis, including fragmentation and condensation of hepatocytes, forming intensely eosinophilic Councilman bodies, which are characteristic of viral hepatitis. Formation of ropelike eosinophilic structures within hepatocytes, called Mallory bodies, is typical of alcoholic hepatitis. Another finding in acute hepatitis is an inflammatory infiltrate in the portal tracts. Chronic hepatitis C is characterized by the formation of lymphoid aggregates in the portal tracts as well as fatty changes in hepatocytes.

This page intentionally left blank ❖ CASE 2 A 30-year-old male banker complains of midepigastric gnawing and boring pain for the last week. The pain is worse at night and is somewhat better immediately after he eats. He has not had any fever, nausea, or vomiting. He takes approximately one 500-mg acetaminophen tablet a week for headaches but does not take any other medications. Upper endoscopy reveals a 2-cm mucosal defect in the antrum of the stomach. There is mild edema in the adjacent mucosa, but there is no thickening of the edges of the ulcer.

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